Lab Information

John Di Battista (PhD)

Associate Investigator
Centre for Translational Biology
Department of Medicine (McGill)

Research Profile

 Fundamental: 75%
 Clinical: 25%
 Epidemiology: 0%
 Evaluation: 0%
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Keywords

Signaling • genetics • proteomics • G protein-coupled receptors (GPCRs) • inflammation

Research Interests

My research focuses on G-protein coupled receptor signaling pathways regulating the inflammatory response.


Team Members

Name Position

Latest Publications

  1. Zriek, F., Di Battista, J. A. & Alaaeddine, N. (2021). Mesenchymal Stromal Cell Secretome: Immunomodulation, Tissue Repair and Effects on Neurodegenerative Conditions. Current stem cell research & therapy, vol. 16, p. 656-669.
  2. Zhao, C., Amiable, N., Laflamme, M., Marsolais, D., Di Battista, J. A., Fernandes, M. J. & Bourgoin, S. G. (2019). Impairment of chemical hypoxia-induced sphingosine kinase-1 expression and activation in rheumatoid arthritis synovial fibroblasts: A signature of exhaustion?. Biochemical pharmacology, vol. 165, p. 249-262.
  3. Gerarduzzi, C. & Di Battista, J. A. (2017). Myofibroblast repair mechanisms post-inflammatory response: a fibrotic perspective. Inflammation research : official journal of the European Histamine Research Society ... [et al.], vol. 66, p. 451-465.
  4. Gerarduzzi, C., He, Q., Zhai, B., Antoniou, J. & Di Battista, J. A. (2017). Prostaglandin E2-Dependent Phosphorylation of RAS Inhibition 1 (RIN1) at Ser 291 and 292 Inhibits Transforming Growth Factor-β-Induced RAS Activation Pathway in Human Synovial Fibroblasts: Role in Cell Migration. Journal of cellular physiology, vol. 232, p. 202-15.
  5. Zhao, C., Moreno-Nieves, U., Di Battista, J. A., Fernandes, M. J., Touaibia, M. & Bourgoin, S. G. (2015). Chemical Hypoxia Brings to Light Altered Autocrine Sphingosine-1-Phosphate Signalling in Rheumatoid Arthritis Synovial Fibroblasts. Mediators of inflammation, vol. 2015, p. 436525.
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